Objective Mice genetically deficient in endothelial nitric oxide synthase (eNOS?/?) are
Objective Mice genetically deficient in endothelial nitric oxide synthase (eNOS?/?) are hypertensive with lower circulating nitrite amounts, indicating the need for constitutively created nitric oxide (NO?) to blood circulation pressure rules and vascular homeostasis. NO? tank. These effects had been abolished from the NOS inhibitor L-NAME and repristinated from the NOS substrate L-Arginine, and had … [Read more…]