Mutations in individual CLC-1 chloride route are from the skeletal muscle
Mutations in individual CLC-1 chloride route are from the skeletal muscle tissue disorder myotonia congenita. the Hsp90 inhibitor 17-allylamino-17-demethoxygeldanamycin (17-AAG) that significantly suppresses cullin 4 appearance. We further verified that cullin 4 may connect to Hsp90 and FKBP8. Our data are in keeping with the theory that FKBP8 and Hsp90 play an important function in … [Read more…]