The cellular mechanisms underlying alcohol addiction are poorly understood. of sEPSC
The cellular mechanisms underlying alcohol addiction are poorly understood. of sEPSC rate of recurrence reaches a maximum with 40mM ethanol and declines with concentrations 80mM ethanol, which is fairly most likely due to D2 receptor activation as raclopride, a D2 receptor blocker, considerably improved 80mM ethanol-induced improvement of sEPSCs. Finally, 6, 7-dinitroquinoxaline-2, 3-dione (DNQX), an … [Read more…]