It’s been widely reported that -amyloid peptide (A) blocks long-term potentiation
It’s been widely reported that -amyloid peptide (A) blocks long-term potentiation (LTP) of hippocampal synapses. during tetanus, hence opposing the induction of synaptic plasticity. Hence, a book pathway by which A can action to modulate neural activity is certainly identified, highly relevant to learning and storage and exactly how it could mediate areas of the … [Read more…]